Metabolism

OVERVIEW

A term 'metabolism' is used to describe chemical reactions in the cells and the organisms, which convert various substances to energy in order to maintain living state. Metabolism is conveniently divided into two categories, catabolism and anabolism. Catabolism is the breakdown of molecules to obtain energy and anabolism is the synthesis of molecules with energy. These metabolic molecules are indispensable to sustain life. Therefore, investigating metabolic pathways is not only of great significance to understand physiological processes but also to treat diseases such as cancer, in which metabolism is different from normal cells. Metabolism antibodies are very important tools in these research processes.

Schematic overview over the main metabolic hubs. Fig.1 Schematic overview over the main metabolic hubs.[2]

METABOLISM ANTIBODY

Metabolism antibody includes protein metabolism antibody, glucose metabolism antibody and liquid metabolism antibody. These antibodies are vital for understanding the homeostatic process of metabolic pathways and numerous diseases resulting from metabolic errors such as obesity, diabetes, lipidemia, hepatic lipidosis, etc.

Mechanism of PCSK9 inhibition with monoclonal antibody. Fig.2 Mechanism of PCSK9 inhibition with monoclonal antibody.[1]

OUR OFFERING

Creative Biolabs supplies antibodies against metabolic markers, metabolites and enzymes. These targets cover a wide area of metabolism research, including basic biological processes (glucose, lipid, protein, and nucleotide metabolism) and metabolic diseases (obesity, diabetes and cancer). These antibodies exhibit high specificity, high affinity, lot to lot uniformity and reproducibility in a range of techniques.

Explore our product range by research area or browse our full metabolism range here. For more detailed information about our antibodies, please feel free to contact us.

REFERENCES

  1. Davidson, M. H.; et al. Emerging low-density lipoprotein therapies: Targeting PCSK9 for low-density lipoprotein reduction. Journal of clinical lipidology. 2013, 7(3), S11-S15.
  2. Hopp, A; et al. Regulation of glucose metabolism by NAD+ and ADP-ribosylation. Cells. 2019, 8(8), 890.
For research use only, not directly for clinical use.
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